1. Testosterone Suspension and Testosterone Base. Testosterone No Ester

    Testosterone is the undisputed king of steroids mainly because it is safe, elicits rapid mass and strength gains while maintaining libido, a sense of well being and energy. It’s not uncommon for a first time user to gain 15-20lbs of LBM in a standard Testosterone cycle. Pure Testosterone comes in a water based aqueous form (Suspension) and also in a solvent/oil based form (Test Base).

    Suspensions have tiny particles that are visible with the naked eye. If left on the shelf for a few days many times the particles will sink to the bottom leaving the clear solvents and water on the top. Depending on the manufacturer, particle sizes vary meaning some Suspension preparations can clog a 22 gauge needle. Ultra micronized Suspension can pass through a 25 gauge needle making injections more comfortable. Shake the suspension preparation vigorously before injecting.

    Testosterone suspension is the most potent form of testosterone because it does not possess an ester. Esters are calculated into the steroid weight therefore esterfied steroids are not a true mg for mg of free hormone. 100mg of suspension is 100mg of free hormone! Enanthate in a solution is only 72mg of free hormone per 100mg. You can see that Suspension is the true king steroid. However because there is no ester many users will inject suspension everyday or even multiple times per day. This is usually reason enough for most people to reject using suspension but it gets worse. Usually suspension is quite painful as well. Combine every day injections with significant pain and most users simply pass on trying suspension at all. Some new science now demonstrates that everyday and even every other day injections are not necessary with Testosterone Suspension.

    What is the real half life of Testosterone Suspension?

    There is no classic half life of aqueous Testosterone Suspension due to the nature of the various suspension particle sizes and the non existence of an ester. In other words we don’t see the same types of decay rates with blood androgen levels in non esterfied preparations that are seen in esterfied preparations. However in March of 2011 there was a pharmacokinetics study done in horses that reported a median terminal half-life of 39 hours with aqueous Testosterone Suspension. The disposition of testosterone from this formulation was characterized by an initial, rapid absorption phase followed by a much more variable secondary absorption phase. There were at least two plasma testosterone concentration peaks. The first peak is almost immediate and the second peak is a whopping 7 days later on average according to the chart in the full study. The study indicates that the initial peak is from the Testosterone formulation solution and the following peak(s) from the solid material in the suspension.

    Basically the solution almost immediately hits the blood stream when injected and then a few days later the solid particles are slowly absorbed by the body causing other peaks in testosterone blood androgen levels.

    So how often should you administer Suspension?

    Based on this science, injecting Suspension every other day or even every three days will maintain high blood androgen levels. The king of steroids has had a time release delay built into it all along and we have the data to prove it. 100mg every other day would be a good starting dose for newer male users. More advanced male users could easily double that dose for very rapid and pronounced LBM gains. Suspension is moderately estrogenic and that effect will be dose dependant. The more you administer the more likely aromatase activity will occur. I would use Nolvadex to lower estrogenic side effects or an aromatase inhibitor.

    Sample 8 week Suspension cycle

    Monday  - 150mg Suspension/20mg Nolvadex
    Tuesday - 20mg Nolvadex
    Wednesday  - 150mg Suspension/20mg Nolvadex
    Thursday -   20mg Nolvadex
    Friday -  150mg Suspension/20mg Nolvadex
    Saturday -  20mg Nolvadex
    Sunday -  20mg Nolvadex

    Nolvadex is used to keep lipids positively influenced for those concerned with cardiovascular health. I have opted for an injection schedule of only three times per week to allow for comfort and because a more frequent schedule is not needed.

    This cycle should produce rapid increases in strength and mass. I would use this cycle during a bulking phase. A more adventuresome user could stack a strong oral like Dianabol or Anadrol with the above cycle at 50mg daily producing an amazing and rapid increase in size and strength if nutrition, training and recovery are dialed in.

    Testosterone Base~Oil/Solvent Based

    Testosterone Base is 100% pure testosterone similar to aqueous Testosterone Suspension however Test Base is technically a solution not a suspension. Test Base contains no visible Testosterone crystals because they are in an oil and solvent solution NOT water. There are no crystals to slowly absorb into the injection site. Therefore when you inject Test Base there is a very rapid increase in blood Testosterone levels that falls off faster than standard aqueous Testosterone suspension. Test Base packs a big instant wallop when injected and is arguably the fastest Testosterone product available today. Another advantage of Test Base is you may use very small gauge needles to inject it. There are no crystals in Test Base to clog the needle so administering with an insulin syringe is an option. Test Base is ideal pre training or for power lifting or strength sports. I recommend Test Base to be administered two hours pre-training to provide increased aggression and power

  2. Halotestin is a good compound for use by athletes

    Unfortunately for bodybuilders, its muscle-building attributes are not as effective. Halotestin doesn’t convert to estrogen in the body, but it is toxic to the liver, so small doses are recommended.

    Halotestin is the Upjohn brand name for the steroid fluoxymesterone. Structurally fluoxymesterone is a derivative of testosterone, differing from our base androgen by three structural alterations (specifically l7alpha-methyl, 11 beta-hydroxy and 9-fluoro group additions). The result is a potent oral anabolic steroid that exhibits extremely strong androgenic properties. This has a lot to due with the fact that it is derived from testosterone, and as such shares important similarities to this hormone. Most importantly, like testosterone, Halotestin appears to be a good substrate for the 5-alpha reductase enzyme. This is evidenced by the fact that a large number of its metabolites are found to be 5-alpha reduced androgens, which coupled with its outward androgenic nature, suggests it is converting to a much more active anabolic steroid in androgen responsive target tissues such as the skin, scalp and prostate.

    The 11 beta-hydroxyl group also inhibits aromatization, making estrogen production impossible with this steroid. Estrogenic side effects such as water retention, fat fain and gynecomastia are therefore not a concern when taking this compound. Strong androgenic side effects are to be expected though, and in many cases are unavoidable. Oily skin and acne a very common for instance, at times requiring sensitive individuals to seek some form of topical or even prescription drug treatment to keep it under control. Hair loss is an additional worry, making Halotestin a poor choice for those with an existing condition. Aggression may also become very pronounced with this drug. This effect is often desired by users looking to “harness” this in order to increase the intensity of workouts or a competition. Clearly Halotestin is a strong androgen, and definitely one female athletes should stay away from. Masculinizing side effects can be intense, and may occur very rapidly with this substance. Even women daring enough to take Dianabol should think twice about this compound, as virilization symptoms are most often permanent.

    Although Halotestin appears to be more androgenic than testosterone, the anabolic effect of it is not very strong. This makes it a great strength drug, but not the best for gaining serious muscle mass. The predominant effect seen when taking Halotestin is a harder, more dense look to the muscles without a notable size increase. It is therefore very useful for athletes in weight-restricted sports like wrestling, powerlifting and boxing. The strength gained from each cycle will not be accompanied by a great weight increase, allowing most competitors to stay within a specified weight range. Halotestin also makes an excellent drug for bodybuilding contest preparation. When the competitor has an acceptably low body fat percentage, the strong androgen level (in absence of excess estrogen) can elicit an extremely hard and defined (“ripped”) look to the muscles. The shift in androgen/estrogen ratio additionally seems to bring about a state in which the body may be more inclined to burn off excess fat and prevent new fat storage. The “hardening” effect of Halotestin would therefore be somewhat similar to that seen with trenbolone, although it will be without the same level of mass gain. Clearly non-aromatizing androgens such as Halotestin and trenbolone can play an important role during contest preparations.

    The main concern with Halotestin is that it can be a very toxic drug. This is due to the fact that fluoxymesterone is a 17 alpha alkylated compound, its structure altered to survive oral administration. l7alpha alkylation can be very harsh to the liver. The possibility of damage is therefore a legitimate concern with Halotestin, especially when used at higher doses or for prolonged periods of time. The total daily dosage is likewise best kept in the range of 20-40mg, used for no longer than 8 weeks. After which an equally long break (at a minimum) should be taken from all c17-AA orals. One should also resist the temptation to stack this drug with other alkylated orals if possible, and instead opt for orals without this alteration or esterified injectable compounds (which will not add to the strain on the liver).

    In cutting phases a mild anabolic such as Deca-Durabolin or Equipoise might prove to be a good addition, as both provide good anabolic effect without excessive estrogen buildup. Here Halotestin will provide a well needed androgenic component, helping to promote a more solid and defined gain in muscle mass than obtained with an anabolic alone. Perhaps Primobolan Depot would even be a better choice, as with such a combination there is no buildup of estrogen (and likewise even less worry of water and fat retention). For mass we could alternately use an injectable testosterone. A mix of 400-800mg Testosterone enanthate and 20-30mg Halotestin for example, should prove to be an exceptional stack for strength and muscle gain. This however would be accompanied by a more significant level of side effects, both compounds exhibiting strong androgenic activity in the body.

    Halotestin also seem to depress endogenous testosterone levels rather quickly with use, despite its complete lack of estrogen conversion. One therefore should consider ancillary drug use at the conclusion of each cycle in order to help restore the normal release of androgens in the body. Using a combination of HCG and Clomid/Nolvadex is of course the best option, the two drugs working well together to restore normal hormonal functioning. Although estrogen is not a problem with Halotestin, the use of an anti-estrogen such as Nolvadex or Clomid is still indicated when discontinuing a cycle. Since HCG stimulates aromatase activity in the Leydig’s cells, here Nolvadex/Clomid help by blocking the activity of any excess estrogen that may be produced. Afterward they will also block the inhibitory effect of endogenous estrogens on the hypothalamus, stimulating the enhanced release of gonadotropins and supporting the normal biosynthesis of testosterone.

  3. Anti-Estrogens and Anabolic Steroids

    Because of their ability to reduce risk of gynecomastia (abnormal growth of breast tissue in males) and enhance recovery of natural testosterone production after a cycle, use of anti-estrogens such as Clomid (Clomiphene citrate) has become popular in bodybuilding. Anti-estrogens also can reduce bloating associated with anabolic steroid use, and may avoid health risks associated with elevated estrogen levels. Medically, the drugs are used not only for treatment of breast cancer but also for improvement of fertility in both men and women, and occasionally for increasing testosterone levels in men such as endurance athletes with low testosterone. There are two categories of anti-estrogens: aromatase inhibitors and receptor blockers. Both shall be considered here.

    As with androgens, where any hormone that has the activity of testosterone is an androgen and therefore all anabolic steroids are androgens, any hormone that has the activity of estradiol, the principal female sex hormone, is an estrogen. The most active natural estrogens in humans are estradiol and estrone. These hormones are related to each other rather similarly to how the andro prohormones are related to each other. Just as androdiol has a hydroxy (or –ol) group at both the 3- and 17- positions, estradiol likewise has a hydroxy group at those positions. Estrone, like androstenedione, has keto (or –one, pronounced “oan”) groups at those positions. Estradiol is the most potent (effective per milligram) of the natural estrogens. It is produced either from testosterone via the aromatase enzyme, or from estrone via the estrogenic 17b-HSD enzyme. Estrone is less potent, but all this means is that one needs more of it to accomplish the same job. It is produced either from androstenedione via aromatase, or from estradiol via the same 17b-HSD enzyme working in reverse. From the standpoint of the bodybuilder using anabolic steroids, if nothing is done about the situation, high estrogen levels can cause gynecomastia, will inhibit natural testosterone production, and will cause bloating. High estrogen levels also make it more difficult to lose fat, and tend to cause female pattern fat distribution even in males. Estradiol also has carcinogenic metabolites, and a liver problem sometimes associated with anabolic steroids use, hepatic cholestasis, is caused not by androgen but by an estrogen metabolite. It is also not unusual for bodybuilders to feel poorly on beginning a cycle of high dose testosterone without antiestrogens, and for this reason many have advocated starting with a low dose and building up. However, I strongly suspect that the real problem is estrogenic effect on mood, and the problem can be avoided with use of an aromatase inhibitor.

    Aromatizable Steroids

    Though most bodybuilders feel they know which anabolic steroids aromatize and which do not, sometimes the beliefs are in error. This is because progestogenic activity (activity like that of progesterone, another female hormone) is easily mistaken for estrogenic activity. Both hormones can cause bloating, and both can cause gyno. So anabolic steroids which are capable of activating not only the androgen receptor but also the progesterone receptor are often mistakenly assumed to aromatize.

    Nandrolone is proven to be a progestin. This fact is of clear importance in bodybuilding, because while moderate Deca-only use actually lowers estrogen levels as a consequence of reducing natural testosterone levels and thus allowing the aromatase enzyme less substrate to work with, Deca nonetheless can cause gyno in some individuals. Furthermore, just as progesterone will to a point increase sex drive in women, and then often decrease it as levels get too high, high levels of progestogenic steroids can kill sex drive in male bodybuilders, though there is a great deal of individual variability as to what is too much. Incidentally, this progestogenic activity also inhibits LH production, and contrary to common belief, even small amounts of Deca are quite inhibitory, approximately as much so as the same amount of testosterone.Well, antiestrogens can do nothing about these side effects of Deca.

    The anabolic steroids where aromatization is of particular concern are testosterone, methandrostenolone (Dianabol), boldenone (Equipoise), and to some extent fluoxymesterone (Halotestin). However the latter is usually used in doses low enough that aromatization is not an issue. Among the prohormones, androstenedione is the principal offender with regard to aromatization, being readily converted to estrone. With androdiol, only that small portion which converts to testosterone can be converted further to estradiol, and that will occur only in the same percentage that other testosterone converts to estradiol. Norandrodiol cannot convert directly to estrogen, and even after conversion to nandrolone is not readily converted to estrogen.

    Norandrostenedione can be converted to estrone by aromatase, but is a very poor substrate for that enzyme. It can actually act as a competitive inhibitor, blocking better substrates such as androstenedione or testosterone. It is possible then, though unproven, that norandrostenedione might have some value as an aromatase inhibitor in bodybuilding. I do think, however, that the pharmaceuticals designed for the purpose should be assumed to be better choices.

  4. Cardiovascular Health and the Anabolic Steroid Using Bodybuilder

    In millennia past and up until these last few decades, bodybuilding was largely viewed as a healthy physical practice by both those who participated in it, as well as the general public. In truth, the original basic tenets of this lifestyle, which involved lifting weights, healthy eating habits, getting plenty of rest, fresh air, and sunshine, were all geared towards improving the health & physical appearance of the individual. The benefits acquired from engaging in such activities are well documented, supporting the claim that the bodybuilding lifestyle was one of health, vitality, and longevity.

    However, over the last few decades, bodybuilding has for many slowly morphed into an endeavor based almost solely on appearance, with any tangible health benefits being more of an afterthought rather than an intended objective. While many of the health benefits related to exercise and nutrition are still present, the world of anabolic steroids has thrust upon the bodybuilding community a new wave of potential health risks. While we have come a long away in our understanding of these drugs and have therefore been able to largely circumvent their side effects, many bodybuilders have left themselves vulnerable to the most serious health risks associated with anabolic steroid use. These are the cardiovascular health risks, such as heart attack & stroke.

    Today, most anabolic steroids related cardiovascular health issues are caused by damaged cholesterol/lipid values, high blood pressure, and elevated hematocrit. Anyone one of them alone can be problematic, but many bodybuilders suffer from all three, vastly compounding the potential for harm. With these side effects having taken up residence in a disturbingly large number of both current and former anabolic steroid users, this subject deserves the attention of every anabolic steroid using bodybuilder. At a minimum, those who reap the benefits of these drugs should demonstrate responsibility in monitoring this vital aspect of their health. An essential part of this process includes getting regular physician monitored blood work, which is necessary for informing us of all relevant health markers and thereby assisting us in taking the proper corrective action.

    While individual anabolic steroid can vary widely in their effects on the human body, some categories of steroids are generally more prone to causing certain side effects. Naturally, steroids have been separated into two primary categories, those of oral (methylated) and injectable anabolic steroids. Of the two, orals tend to present the greatest degree of risk to our cardiovascular health, as their ability to rapidly and significantly affect the user’s cholesterol profile cannot be understated. With this said, oral anabolic steroids also have a solid place in the bodybuilders repertoire and when used responsibly, they have the potential to greatly assist the bodybuilder in achieving his goals. Injectables can also cause adverse alterations to the bodybuilders cholesterol profile, but as a whole, they’re less deleterious in this regard.

    With injectables, total dose, type of drug used, and whether or not the drug aromatizes, will all play a role in determining its ability to negatively impact cholesterol values. Typically, injectable anabolic steroids which do not aromatize, are used in higher dosages, and which display a more potent androgenic component, will have greater potential to negatively affect our lipid profile than a low-dosed, aromatizing anabolic. Two contrasting examples would be Trenbolone and Nandrolone. Trenbolone is known for being a less forgiving anabolic steroids in many ways, and cholesterol is no exception. Many bodybuilders taking this drug notice adverse changes to their cholesterol levels, while Nandrolone is much less likely to be problematic in this area. While these characteristics do not always accurately reflect which steroids will be the greatest cholesterol offenders, they are often an accurate indicator.

    Cholesterol/lipids play a significant role in the bigger picture, but it is by no means the sole cause of cardiovascular health problems in anabolic steroids using bodybuilders. Blood pressure is another contributing factor, which should be regularly monitored by the individual. Unlike cholesterol/lipids, which require blood work in order to get any definitive answers, blood pressure can be checked with a simple device right in the privacy of your own home. For bodybuilders who do not have the equipment needed to check their own BP, many pharmacies or similar places of business will provide a self-automated blood pressure machine, which can be used free of charge. Generally, a BP reading of 120/80 is considered ideal, but anywhere below 140/90 is considered OK. Once a BB’rs begins to get above that range, he should begin taking steps to help restore a more normal reading.

    There is great variability among anabolic steroids when it comes to increasing blood pressure. Generally, those anabolic steroids which cause a large amount of water retention and result in quick mass gains are the most notorious for elevating BP. Steroids which result in lean, water free increases in muscle tissue are less likely to be problematic in this area, although there are exceptions to the rule. Unlike cholesterol, in which oral anabolic steroids present the largest degree of health risk, methylation does not appear to be a relevant factor in determining whether or not a steroid is likely to elevate an individual’s blood pressure. When determining which anabolic steroids are most likely to cause this effect, each steroid must be evaluated on a case by case basis. Chronically elevated BP, like cholesterol, can do long-term damage, resulting in cardiovascular consequences years after the bodybuilder has discontinued using anabolic steroids. This should behoove all bodybuilders to make wise short-term decisions, so that long-term good health becomes a reality.

    Hematocrit refers to the volume percentage of red blood cells in the blood. When a bodybuilders hematocrit starts to rise outside of the normal range, it has the effect of thickening the blood. The thicker the blood, the harder the heart has to work to pump blood throughout the body. This increases stress on the heart similar to how high blood pressure might increase stress on the heart. High hematocrit also increases the possibility of stroke through an increased risk or forming blood clots. As a whole, steroids are well known to increase RBC, although some tend to do this more proficiently than others. At one point, Anadrol was used medically for this purpose, prior to the advent of prescription EPO. In many ways increased RBC can be beneficial, imparting increased endurance, vascularity, and greater muscle pumps to the user, but when it climbs too high, it becomes another contributing factor to cardiovascular health risk.

    Today, many bodybuilders suffer from the cardiovascular health issues mentioned above, but few monitor this area of their health on any type of a regular basis, let alone take steps to minimize the occurrence of damage. The symptoms which manifest themselves in the presence of poor cardiovascular health often go unnoticed, making heart attack the #1 killer of American men today. Anabolic steroids using bodybuilders willingly engage in a practice, which has been conclusively shown to elevate their degree of risk substantially, yet many bodybuilders assume a lax attitude in the area of preventative care. Through continuing education, we can continue to reverse this trend, even as some industry voices have already begun to make inroads into this destructive mind-set.

    Fortunately, for the bodybuilder who seeks to adopt an attitude of responsibility in terms of cardiovascular health care, there is an abundance of knowledge available on the subject.

  5. Gynecomastia: Causes.

    Out of all the side effects a anabolic steroid user could potentially experience, gynecomastia is one of the worst and it certainly tops the list when it comes to cosmetic side effects. Being heavily associated with womanhood, the very nature of this side effect is an affront to our masculinity. Not only is it embarrassing, but in some cases it can destroy the entire appearance of one’s physique. We have all seen pictures of the more extreme cases, where the bodybuilders literally looks like he has grown a small pair of tits on an otherwise normal body. The severity of this condition can range from only slight swelling, which is often imperceptible from a visual standpoint, to the more extreme cases, as mentioned above Fortunately, gynecomastia does not develop over night and its progression is easily halted and reversed if the proper steps are taken in a timely fashion. There really isn’t any good excuse for a anabolic steroid user to experience permanent, visible breast tissue growth. Those that do either don’t care or rather (and much more likely), they weren’t prepared and/or educated to deal with this side effect ahead of time. While there are numerous potential causes of this condition, the bodybuilders generally only has to worry about a few of them and the available treatment options are simple in their application. Through minimal self-education and a small financial investment we have all the tools we need to keep this side effect at bay.

    Why are my nipples sore?

    The most important factor in the prevention of gynecomastia is knowing how it occurs in the first place. There are 4 types of steroids which can cause gynecomastia, the most prominent of which are the aromatizable steroids. Aromatizable steroids are those anabolic steroids which are capable of converting to estrogen. This includes many anabolic steroids, such as Testosterone, Methyltestosterone, Methandrostanolone (Dianabol), and to a lesser degree, Nandrolone (Deca/NPP), and Boldenone (EQ). This conversion process is initiated when aromatizable anabolic steroids interact with aromatase, which is an enzyme necessary for the biosynthesis of estrogen. See below for a depiction of this interaction:

    When low dosages of these anabolic steroids are administered, the degree of conversion is not sufficient to cause this side effect. However, this threshold is regularly crossed by bodybuilders who utilize dosages well in excess of those required to avoid estrogenic side effects. The problem with being able to pin-point the dose at which this side effect occurs is that not all individuals are affected equally by the same dose. Some can get away with using fairly large dosages of highly aromatizing drugs, such as testosterone, while others seem to develop problems even with minimal use. I know a powerlifter who was able to use a full gram of pharmaceutical-grade testosterone per week without experiencing any gyno symptoms, while other people have encountered early signs of gynecomastia at only 250 mg weekly. For this reason, assumptions should not be made beforehand regarding personal tolerance. When it comes to learning your limit, real-world experience must be your teacher. However, if I had to guess, I would say that most individuals will begin to experience the early stages of gyno by the time they reach 400-500 mg per week when using testosterone, assuming preventative action is not taken.

    The 2nd type of anabolic steroid which can potentially cause gyno are those which appear to exhibit inherent estrogenic activity, despite lacking the ability to aromatize. One example would be Oxymetholone (Anadrol). This anabolic steroid is not capable of converting to estrogen to any degree, yet it causes gyno in a moderate percentage of users. The most plausible explanation for this side effect is that the Anadrol molecule itself demonstrates estrogenic activity, likely by attaching directly to the estrogen receptor. While we cannot yet conclusively state this to be fact, it seems more probable than the other theories which have been put forward over the years.

    The 3rd type of anabolic steroids associated with the development of gynecomastia are the progestin-based steroids, which promote their effects through direct stimulation of the progesterone receptor. Two examples which fit this description would be Trenbolone and Nandrolone. However, these drugs differ from the previously mentioned anabolic steroids in that they rarely cause gyno on their own. Typically, they require the presence of estrogen (usually at above normal levels) in order to have an impact on the growth of breast tissue. In essence, their progestagenic effects tend to exacerbate the effect of estrogen, thereby acting more as a contributor rather than the primary offender.

    The last category of anabolic steroids with a history of exhibiting this side effect are those which result in estrogen-rebound. Anticipating the effects of these drugs can be difficult, as they are much less predictable in their behavior. Not only does personal response vary tremendously between individuals, but there is often a lack of consistency even among the same individual. For example, a particular anabolic steroid may be employed multiple times without incident, only to cause troubling gyno symptoms under nearly identical circumstances at a later date.

    We’ve looked at particular types of anabolic steroids and how they work to cause gyno, but there are other causes which should not be overlooked, one of which is an out of balance androgen to estrogen ratio. DHT itself, as well as many DHT derivatives, possess natural anti-estrogenic activity. Therefore, even in the face of stable estrogen levels, a reduction in these hormones may lead to the appearance of estrogenic side effects. In turn, an increase in DHT or its related metabolites will enable an individual to more effectively deal with increased levels of estrogen. This is why a anabolic steroid user is much more likely to encounter estrogenic side effects using Testosterone alone, compared to a combination of Testosterone and Drostanolone (Masteron), which was developed specifically for the management of female breast cancer.

    The last cause of gyno in the anabolic steroid user is prolactinemia, which is an elevation of prolactin levels outside of the normally recognized limits. The most common cause of prolactinemia in the general population is prolactinoma (tumor of the pituitary gland), although this condition can be caused by administering prolactin elevating anabolic steroids, such as Trenbolone and Nandrolone. However, just like progesterone, prolactin rarely causes gyno by itself. This is not because prolactin is not able to accomplish this. Rather, anabolic steroids generally aren’t able to elicit elevations in prolactin adequate for causation. Most of the time, prolactin acts to exacerbate the effects of estrogen (similar to progesterone), making it a contributing factor and not the primary cause in the large majority of cases. Still, there have been instances in which these steroids were able to cause gyno and/or lactation independent of aromatizable anabolic steroids. I want to emphasize the fact that lactation rarely occurs in steroid users, but when it does, it is almost always attributable to excessive dosing with the offending steroids, in combination with poor personal response.

  6. Nandrolone Beats Testosterone for Weight Gain


    Nearly everyone who has used both Testosterone and  Nandrolone decanoate (Deca) will tell you that, at an equal dose, Testosterone will build more muscle and strength. Despite this commonly accepted wisdom, Nandrolone decanoate boasts a 25% higher anabolic rating than testosterone, as measured by Vida (on rodents). And now, a human study performed on AIDS patients, has also suggested that Nandrolone decanoate is about a quarter (*give or take) more anabolic than Testosterone (enanthate). And guess what else? Quality of life markers also showed a greater improvement with Nandrolone decanoate over Testosterone.

    But remember, this study wasn’t performed on healthy, weight-training, athletes…it was performed on people with an autoimmune disease. So why do healthy people seem to get more gains on testosterone than nandrolone, while it appears to be the opposite in those with a viral/wasting condition? I think I have an explanation:

    During the luteal phase of menses (*yes, I’m talking about the menstrual cycle here), women experience a variety of effects on their immune system, including heightened TH1 activity. TH1 cytokines produce proinflammatory responses responsible for killing intracellular parasites and for perpetuating autoimmune responses. It’s no surprise then, that women with autoimmune diseases find that the severity of their symptoms will decrease during this phase – which is marked by an increase in progesterone…

    Which brings us to the fact that Nandrolone decanoate is derived from progesterone, which gives us good cause for thinking that this particular steroid will have an advantage over testosterone in those with an autoimmune disease. So while this study is pretty cool, and shows that anabolic steroids can have beneficial effects in the treatment of wasting diseases, it doesn’t tell us that the average healthy athlete will post more gains with nandrolone over testosterone. Both are great steroids, but in this case, I need to defer to my own experience plus the collective wisdom of literally hundreds of thousands of athletes, and say that test will put more weight on the average athlete than deca.

    And you can always use both…

  7. Testosterone Replacement Therapy

    Testosterone is responsible for normal growth and development of male sex organs and maintenance of secondary sex characteristics. It is the primary androgenic hormone, and its production and secretion are the end product of a series of hormonal interactions. Gonadotropin-releasing hormone (GnRH) is secreted by the hypothalamus and controls the pulsatile secretion of luteinizing hormone (LH) and follicle-stimulating hormone (FSH) by the anterior pituitary. Luteinizing hormone regulates the production and secretion of testosterone by the Leydig cells of the testes, and FSH stimulates spermatogenesis.

    When the testes fail to produce normal levels of testosterone, testosterone deficiency results. Hypergonadotropic hypogonadism is caused by primary testicular failure. Testosterone levels are low and pituitary gonadotropins are elevated. In secondary, or hypogonadotropic hypogonadism, there is inadequate secretion of pituitary gonadotropins. In addition to a low testosterone level, LH and FSH levels are low or low-normal. While pre-pubertal hypogonadism is generally characterized by infantile genitalia and lack of virilization, the development of hypogonadism after puberty frequently results in complaints such as diminished libido, erectile dysfunction, infertility, gynecomastia, impaired masculinization, changes in body composition, reductions in body and facial hair, and osteoporosis. In addition to these complaints, mood inventory scores indicate that hypogonadal men report levels of anger, confusion, depression, and fatigue that are significantly higher than those reported by men with normal testosterone levels.

    Men with primary hypogonadism (congenital or acquired) or hypogonadotropic hypogonadism are candidates for testosterone replacement therapy, and there are now a variety of products available to treat these disorders. Successful management of testosterone replacement therapy requires appropriate evaluation and an understanding of the benefits and risks of treatment.

    Diagnosis of Testosterone Deficiency

    Given the variety of causes of testosterone deficiency, a medical and medication history, physical exam, and directed laboratory evaluation are imperative. The medical history should include questions regarding developmental abnormalities at birth, the rate and extent of virilization at the time of puberty, and the current status of sexual function and secondary sexual characteristics, such as beard growth, muscular strength, and energy level. Hypogonadal men have statistically significant reductions in the incidence of nocturnal erections, the degree of penile rigidity during erection, and the frequency of sexual thoughts, feelings of desire, and sexual fantasies. Alterations in body composition, including increases in percent body fat, changes in adipose tissue distribution, and reduction in muscle mass, are frequently seen in hypogonadal men. Spinal trabecular bone density is also decreased in men with hypogonadotropic hypogonadism and hip fractures are more common in hypogonadal men than in normal men.

    Initially, hormonal screening is limited to measurement of total serum testosterone, which is obtained in the morning. When the total testosterone level is low and/or the patient complains of reduced libido, a serum prolactin level should also be measured. A high serum prolactin level may indicate pituitary dysfunction and may require consultation with an endocrinologist. Serum LH levels are measured when serum prolactin levels are normal or low to help differentiate intrinsic testicular failure from a pituitary or hypothalamic abnormality. LH is usually high in patients with primary testicular disease. When the serum testosterone level is low and LH is elevated, testosterone replacement therapy is warranted.

    Testosterone Replacement Therapy

    Testosterone replacement should in theory approximate the natural, endogenous production of the hormone. The average male produces 4-7 mg of testosterone per day in a circadian pattern, with maximal plasma levels attained in early morning and minimal levels in the evening. However, the subtleties of pulsatile and diurnal rhythms are potentially difficult to imitate, and evidence suggests that different dose response curves exist for different androgen-dependent functions. The clinical rationale for treatment of testosterone deficiency may include:

    • stabilizing or increasing bone density 
    • enhancing body composition by increasing muscle strength and reducing adipose 
    • improving energy and mood 
    • maintaining or restoring secondary sexual characteristics, libido and erectile function

    Types of Testosterone Replacement Therapy

    Ideal testosterone replacement therapy produces and maintains physiologic serum concentrations of the hormone and its active metabolites without significant side effects or safety concerns. Several different types of testosterone replacement are currently marketed, including tablets, injectables, and transdermal systems.

    Oral agents

    Although elevations in liver function tests and abnormalities at liver scan and biopsy are relatively common in patients receiving oral testosterone,10 these preparations still constitute roughly a third of the testosterone prescriptions filled in the United States. Both modified and unmodified oral testosterone preparations are available. Unmodified testosterone is rapidly absorbed by the liver, making satisfactory serum concentrations difficult to achieve. Modified 17-alpha alkyltestosterones, such as methyltestosterone or fluoxymesterone, also require relatively large doses that must be taken several times a day.

    Intramuscular injection

    Testosterone cypionate and enanthate are frequently used parenteral preparations that provide a safe means of hormone replacement in hypogonadal men. Testosterone is esterified to inhibit degradation and to make it soluble in oil-based injection vehicles that retain the drug in muscle tissue. In men 20-50 years of age, an intramuscular injection of 200 to 300 mg testosterone enanthate is generally sufficient to produce serum testosterone levels that are supranormal initially and fall into the normal ranges over the next 14 days. Fluctuations in testosterone levels may yield variations in libido, sexual function, energy, and mood. Some patients may be inconvenienced by the need for frequent testosterone injections. Increasing the dose to 300 to 400 mg may allow for maintenance of eugonadal levels of serum testosterone for up to three weeks, but higher doses will not lengthen the eugonadal period.

    Transdermal systems

    Currently, three testosterone transdermal systems are marketed: a system applied to the scrotum that has no permeation enhancers  and two systems that contain permeation enhancers for application to appendage or torso skin. Scrotal patches produce high levels of circulating dihydrotestosterone (DHT) due to the high 5-alpha-reductase enzyme activity of scrotal skin.

    Clinical studies of transdermal systems demonstrate their efficacy in providing adequate testosterone replacement therapy.Skin irritation may be associated with the use of transdermal systems; however, Testoderm and Testoderm TTS caused significantly less topical skin irritation than Androderm in two separate clinical studies.

    Benefits of Testosterone Replacement Therapy

    A number of benefits of testosterone replacement therapy have been demonstrated, including effects on mood, energy levels, and libido. Long-term follow-up of testosterone replacement in hypogonadal males and a control group indicates that self-assessment of libido was significantly higher (p<0.0001) in the testosterone-treated group. Testosterone replacement has also been shown to enhance libido and the frequency of sexual acts and sleep-related erections. Transdermal testosterone replacement therapy, in particular, has been linked to positive effects on fatigue, mood, and sexual function, as well as significant increases in sexual activity. More specifically, testosterone replacement therapy has been shown to improve positive mood parameters, such as feelings of wellness and friendliness, while reducing negative mood parameters, such as anger, nervousness, and irritability. Testosterone replacement is an effective treatment for some depressive symptoms in hypogonadal men and may effectively augment treatment in selective serotonin reuptake inhibitor (SSRI)-refractory major depression. Relative to eugonadal men, hypogonadal men in one study were impaired in their verbal fluency and showed improvement in verbal fluency following testosterone replacement therapy.

    Testosterone replacement therapy is also associated with potentially positive changes in body composition. In hypogonadal men, testosterone replacement therapy has demonstrated a number of effects, including an increase in lean body mass and decrease in body fat, an increase in weight, and increases in muscle size. Parenteral testosterone replacement in hypogonadal men resulted in improved strength and increased hemoglobin compared to controls. In another study by Urban and colleagues, testosterone administration also increased skeletal muscle protein synthesis and strength in elderly men. Testosterone replacement with transdermal testosterone delivery systems in HIV-infected men with low testosterone levels has been associated with statistically significant gains in lean body mass (p=0.02), increased red cell counts, and improvements in emotional distress. Transdermal testosterone has also been administered to HIV-positive women, yielding positive trends in weight gain and quality of life.

    Improvements in bone density have also been shown with testosterone replacement therapy. Increases in spinal bone density have been realized in hypogonadal men, with most treated men maintaining bone density above the fracture threshold. Testosterone replacement in hypogonadal men improves both trabecular and cortical bone mineral density of the spine, independent of age and type of hypogonadism.In addition, a significant increase in paraspinal muscle area has been observed, emphasizing the clinical benefit of adequate replacement therapy for the physical fitness of hypogonadal men. 

    Contraindications to Testosterone Replacement Therapy

    Testosterone replacement is contraindicated in men with carcinoma of the breast or known or suspected carcinoma of the prostate, as it may cause rapid growth of these tumors. Hormone therapy is also inappropriate in men with severe benign prostatic hypertrophy (BPH)-related bladder outlet obstruction. Use of testosterone to improve athletic performance or correct short stature is potentially dangerous and inappropriate. In addition, the hormone does not correct ambiguous genitalia resulting from androgen deficiency during fetal development and should not be administered at dosages high enough to inhibit spermatogenesis.

    Safety Issues with Testosterone Replacement

    Although testosterone replacement may be indicated in the aging male with documented hypogonadism, this hormone should not be administered with the intent of reversing the aging process in men with normal testosterone levels. Testosterone replacement therapy may be associated with azoospermia, lipid abnormalities, polycythemia, sleep apnea, and the potential for prostate changes.


    The administration of exogenous testosterone as a means of male contraception is under study.  In these men, azoospermia usually results within approximately 10 weeks of beginning therapy. Rebound of the sperm count to baseline levels occurs within six to 18 months of cessation, and subsequent fertility has been demonstrated.

    Lipid Abnormalities

    Physiologic testosterone replacement is known to reduce total cholesterol, low density lipoprotein (LDL), and high density lipoprotein (HDL) levels, but the extent to which these parameters are affected by treatment varies considerably between studies. While reductions in HDL did not reach significance in a study by Tenover and testosterone replacement was not associated with unfavorable changes in lipid profiles in a separate study by Tan and colleagues,research by Anderson and colleagues suggests testosterone replacement therapy may result in significant reductions in HDL and elevations in blood viscosity. Some authorities recommend that lipid values be followed closely in men receiving testosterone replacement therapy.

    Polycythemia and Sleep Apnea

    Polycythemia has been associated with testosterone replacement therapy and is correlated with elevated bioavailable testosterone and estradiol levels. Physiologic replacement with transdermal testosterone, however, resulted in fewer cases of polycythemia than replacement with testosterone enanthate injections. Although the mechanism is unclear, testosterone replacement therapy may also cause or worsen obstructive sleep apnea.

    Prostate Changes

    Although PSA is not specific for prostate cancer, it is a good surrogate for judging the effects of androgens on the prostate. In one study of testosterone-treated men, PSA rose to normal levels but no higher than in the controls, leading the authors to conclude that testosterone-induced prostate growth should not preclude hypogonadal men from testosterone replacement therapy. Indeed, another study indicates that even men who achieved supraphysiologic levels of serum testosterone had no significant changes in PSA levels.

    In a different evaluation, hypogonadal men with normal pretreatment DRE and serum PSA levels who were treated with parenteral testosterone replacement showed no abnormal alterations in PSA or PSA velocity. The authors concluded that any significant increases in these parameters should not be attributed to testosterone replacement therapy and should be evaluated. The effects of transdermal testosterone replacement on prostate size and PSA levels in hypogonadal men have also been evaluated. Prostate size during therapy with transdermal testosterone was comparable to that reported in normal men, and PSA levels were within the normal range.

    Prostate Cancer

    There appears to be little association between testosterone replacement therapy and the development of prostate cancer. The etiology of prostate cancer is apparently multifactorial, and dietary, geographic, genetic, and other influences are all thought to play a role in the development of the disease. Recent studies indicate that testosterone levels have no apparent systematic relationship to the incidence of prostate cancer.

  8. Tamoxifen Citrate

    Tamoxifen Citrate represent Selective estrogen receptor modulator (SERM). This drug is widely used in medicine world as a treatment against breast cancer.

    Instead of the fact that Tamoxifen Citrate is SERM, bodybuilders take it also as an anti-estrogen and as a triphenylethylene. Selective estrogen receptor modulators act as either estrogen receptor agonists or antagonists in a tissue-selective manner. The main reason why it is used by sportsmen, is the fact that it help to prevent gynocomastia. Tamoxifen Citrate does this by competing for the receptor site in breast tissue, and binding to it. That’s mean that tamoxifen act as a estrogen receptor blockade of breast tissue.

    Those bodybuilders/athletes who take Tamoxifen Citrate as a anti-estrogen supplement must remember that it is not the most efficient ancillary compound, but it is considering to be the safest. Tamoxifen Citrate doesn’t actually reduce estrogen in human body, it rather blocks estrogen from estrogen receptors and, in those tissues where it is an antagonist, causes the receptor to do nothing.

    The usage of Tamoxifen Citrate has positive effects on immune system functionality as well as on lipid profile, on both HDL and LDL. Tamoxifen Citrate is often chosen in post cycle therapy. After taking this drug hypogonadic and infertile men saw significant increase in the serum levels of LH, FSH, as well as testosterone levels. In fact, 20mgs of Tamoxifen can possibly raise testosterone levels approximately 150%.

    Female bodybuilders/athletes usually take Tamoxifen Citrate in order to reduce the effect of their own endogenous estrogens. This can lower body fat concentrations, especially in stubborn areas like the hips and thighs.

    Side effects that can occur are: in females – hot flashes, menstrual irregularities and a variety of complications with the reproductive system; in males – reduced gains.

  9. Ending a steroid cycle

    What is a steroid cycle?

    • This is a cycle that refers to the frequency or the schedule to which a given individual uses steroids. The users of these steroids use them at different intervals periodically at a given time.
    • When the steroids are being used, there is a timeline that is often identified as the “On-cycle” but when they are not being used then the period is identified as “Off-cycle”. During the on-cycle period, there are many options to choose from.
    • Another option that is taken up during the on-cycle phase is also the steroid stacks. For those that do not know what this is, steroid stacks are the combinations of anabolic steroids being used during a steroid cycle all together with non-steroids that are a part of this process.
    • Some of these steroid stacks are like thyroid hormones, peptide hormones, anabolic steroids amongst others. For each choice there is a difference in steroid cycle compared to the others.

    How to cycle off steroids cautiously

    • As a steroid user, it is important that before you start a cycle you should widely do your research on the steroids you use and you should not at any given time overlook the details that form a vital part of your steroid. 
    • If you can carefully plan for your cycle then you shall have saved yourself from the trouble of suffering from the side effects after your subscription to a certain drug. The bad side effects are as an effect of a poorly planned cycle.
    • During the cycle, one should use an anti-estrogen which assists in the control of estrogen related water retention and also gynecomastia.

    The end of a cycle

    • After one completes his or her cycle, there should be the administering of an anti-estrogen which is what should be used in the recovery period. This is basically because of the fact that the testosterone levels are low during this period and the estrogen hormonal levels go up within the body.
    • If an anti-estrogen is administered, you can therefore lower your estrogen levels so that you can prevent side effects such as water retention, acne and even depression amongst many others.
    • A drug that could also assist you in increasing your testosterone levels to the expected amount should also be used during the same time. This practice can help you maintain some of the results of the steroid to your body if not most of them as you also avoid the disadvantages that come with cycling off.

    What substances and methods are used during this process?

    • Anti-Estrogens
    • Drugs that increase testosterone levels
    • Post cycle regiments that minimize side effects.

    Kinds of Anti-Estrogens available

    There are a number of Anti-Estrogens available such as:

    • Arimidex used when on anabolic steroids to prevent water retention.
    • Femara for estrogen control
    • Nolvadex which is good for preventing gynecomastia
    • Proviron which is used as an anti-estrogen during a cycle.

    Examples of testosterone boosters

    Some of the substances used in the increase of testosterone are like:

    • Clomid- which is a drug used at the end of a cycle as it can bring the natural testosterone levels back to normal. It is also used for about 3 weeks which are immediately after the cycle ends.
    • HCG- a drug used mostly by the male bodybuilders, this unique drug must be used with other drugs such as Nolvadex. This is also a substance that should not be used for more than about 4 weeks and high doses risk desensitizing your testicles!

    Many guys recommend the use of an anti-estrogen during the whole cycle which can be heightened during the post cycle time for a bit of positive effects.

    There are also some post cycle recovery methods used during this period. These are:

    • The moderate cycle which lasts for about 6 to 8 weeks? This is a regiment which also includes an anti-estrogen substance and Clomid which is similar to HCG. There are also 3 effective means of having the best effects ever after using Clomid.
    • Then there is the long cycle which is meant to last 4 months or more.

    It is important to note that no cycle should start without the existence of a detailed anti-estrogen package and one should also have post cycle drugs before the cycle is started just in case you are unable to come off once.

    It sounds great to take these measures before they bring you to a harsh world of reality where the side effects have happened. It is therefore possible to have more effective cycles as you reduce the side effects that can be very common and related to the use of steroids. You can take in our advice if you want to stand out.

  10. Anabolic Cycles - how to stack anabolic steroids

    With the increase in the use of anabolics, it is very important to understand and choose the right anabolic cycles to achieve the required goals. In the start, some physicians worked out on making the anabolic steroid cycles for many people including the sportspersons, especially the athletes so that these people get maximum benefits from them. Techniques were starting to get sorted out to minimize the negative effects but unfortunately this process keeps on getting done on the individual basis and that is the reason users had to develop their own cycles according to their specific requirements. There are many steroid cycles introduced and some are more effective than others are, and have given much better results. When you want to use steroids, the first thing that is of utmost importance is to make the right kind of selection for the steroid that satisfies your requirements.

    Selection Criteria for Anabolic Steroids

    The selection criteria for the anabolics differ for different persons according to their own specific needs. It is commonly seen that people normally gets confused while choosing the right kind of anabolics for themselves because anabolic steroids contains various kinds of medications. All the medications or drugs have their own characteristics, which are according to the specific needs of the persons. Some drugs are known for their low androgenic effects and their side effects are few in both the women as well as children. On the other hand, some anabolics are renowned to give a better functioning related to the sexuality of the men. The way of administration of anabolics also differs as some are used as injections while others are being handled orally. It is important to note that the limitations and side effects must be considered and sorted out well before use. It is very important and interesting to note that anabolic steroids deals and activate the same receptor that is cellular one, and all the anabolic steroids have the same properties related to the anabolization of proteins. However, many or few properties may differ and this helps in gaining the desired objective in a perfect way.

    Perfect Muscle Mass and Strength

    Anabolic steroids are normally used to gain muscle mass and to improve the strength of the body. However, it is important to note that there are certain advantages of one anabolic steroid than others, which makes it better. It is often found that the quantity and the quality of the muscle may be different using different kinds of anabolic steroids. Anabolic steroids can frequently increase the total muscle size drastically. There are some drugs which are thought to have no significant estrogenicity effect and that is the reason their size gains are less as compared to the others. The most popular steroid drugs are of two main categories namely, mass (bulking) and the lean mass.

    Starting Anabolics Cycle

    Normally in the start, steroid cycle is used with single anabolic steroid. People want to gain muscle mass and that is the reason they start taking steroids. Doctors advise the first time users to use the injections and they normally choose the oral compound for the sake of ease in their anabolics cycle. Stanozolol is among the anabolic steroids that are preferred by the people for gaining the lean mass.

    Cycle Duration

    When you or your doctor chooses the required usage, then the main thing to consider is the cycle duration and normally anabolic steroid cycle is continued for six to eight weeks. During this time, the muscle rate increases. After that, it slows down significantly. Dosages can be treated as per requirements and if specific need arises, then you can increase the dosage but a consultation must be done with the doctor in that case. It is advisable not to extend the anabolic steroid cycle more than eight weeks at a time; and after some gap, the steroid cycle can be repeated to have maximum benefits but that must be done with the consultation of the specialist.

    Recovery after Cycle Off

    The period after the anabolic cycle is very important and it can involve the state of hypogonadism and due to its result protein catabolism can arise. Proper care must be taken to minimize the loss of muscle gained during the anabolic cycle. Some doctors advise to start the hormonal program in which a person can use the HCG and clomiphene for better results. Some people also use the strategy of reducing the dose of the anabolics rather than leaving it completely at once and that thing is called tapering.

    Stacking of Anabolic Steroids

    When a person gets used to the use of one anabolic steroid, he/she can start the use of more than one anabolic steroid. The technique is called stacking and it is in hot use nowadays. Stacking has gained its popularity normally with the body builders and they gain physical development more effectively that is not possible with the use of single anabolic steroid. Stacking became popular in the 1960s for the very first time. It is very effective especially when an anabolic androgen stack allowed people to use a high dose with a tolerable strength of single androgen. Stacking must be done with drugs that suits to the needs of the body of the specific person; otherwise, it is better to use the single anabolic steroid in a periodic manner to obtain the goals like mass maintenance and performance.

    For great definition and muscularity, during diat, Halotestin and Trenbolone (acetate, enanthate or hexahydrobenzylcarbonate) which are strong and non-aromatizing anabolics can be used for providing arl, enhanced level of hardness and density to the muscles.

    For bulk cycles, being a moderately strong muscle builder, nandrolone decanoate can be used with the classic ” Deca-Durabolin and D-bol” stack (usually 200-400 mg of nandrolone decanoate per week and 20-50 mg of Dianabol per day) has been a bodybuilding basic, the “old school” for decades, and always provide excellent muscle growth.

    For “brutal mass” cycles, stronger anabolic steroids such as Oxymetholone (Anapolon) or any Testosterone (Testosterone Depot,  Testosterone propionate, Sustanone ) could be stacked with Deca-Durabolin, producing greater results, but with more water retention.

  11. Nolvadex - best antiestrogen for steroid cycles

    If you are about to undergo a steroid cycle of 6-8 weeks and have a history of excess estrogen formation after using harsh or aromatizable steroids, Nolvadex is one name on which you can rely without any second thoughts.

    Nolvadex, which is also known as Tamoxifen Citrate, is the best antiestrogen for professional sportsmen using anabolic steroids and performance enhancing drugs when it comes to reducing or eliminating the formation of estrogens without compromising on benefits of steroids. Since some of the most popular anabolic steroids are derivatives of testosterone and leads to excess estrogen formation, ready supply of Nolvadex is an absolute must before the steroid cycle comes to an end.

    One of the best things about this antiestrogen, Nolvadex, is that it doesn’t inhibit all estrogens but only the harmful estrogens. This unique property helps sportsmen on steroids to benefit from positive aspects of estrogens and inhibit harmful estrogens from causing any damage.

    The active ingredient of Nolvadex is tamoxifen citrate and the inactive ingredients are magnesium stearate, mannitol, starch, and carboxymethylcellulose calcium. This antiestrogen is also effective in treating women patients with breast cancer.

    When used in doses of 20-40 mg per day, Nolvadex considerably reduce the risk of excess estrogen formation so that steroid users can extract the best out of steroids without coming in close contact with steroid side effects. The doses of Nolvadex should not be altered without medical recommendation else it could lead to side effects such as depression, dizziness, headache, and hot flashes.

  12. High intensity or volume training

    The argument still goes on and on. The two training camps, volume training and high intensity training, first became delineated when back in the 1970’s Arnold Schwarzenegger was recommending 20 sets per bodypart for maximum growth while Mike Mentzer recommend only one set to failure for each bodypart. That battle over bodybuilding training theory is still raging today. But who is right and who is wrong? To find out, let’s explore training intensity for a moment.

    Weight training intensity is: the amount of work/effort per unit of time. This means if you’re used to performing 10 sets in 20 minutes, if you increase your sets to 15 in the same 20 minutes, then you’ve increased your intensity by 50% as long as you lifting the same weights. Using this same example, if you increase the amount of weight (more effort) then you’ve also increased training intensity. Finally, if you use the same weight, the same number of sets in the same amount of time, but you push each set with more repetitions, then you’ve also increased intensity.

    You can readily see that each of these methods has it’s limits. One practical point to make clear is that you should attempt to perform the most amount of work and generate the greatest effort in the least amount of time. We recommend the ideal training time to be limited to 45 minutes based on blood tests indicating optimum hormonal peaks.

    Idealistically speaking, after you’ve warmed up, you can generate the most intensity and effort from you first set than you can from your tenth. This means two things. Perform your first set carefully because this is when you’re strongest and most likely to injure yourself. Severe injuries rarely happen on the final set because your ability to generate force is at it’s lowest level. When you injure yourself on that last set, it’s usually mental fatigue more then muscular fatigue. Secondly, work your weakest or high priority bodyparts first, when you’re strongest. Another point to make is not everyone can generate maximum effort on that very first work set. Some trainees take several sets to “get into the rhythm” which works perfectly fine. Pyramiding up in weight and effort works great, for many, as long as there’s an on-going push to reach peak performance as quickly as possible.

    So, how many sets do you need to stimulate growth? Isn’t this the question you really want answered? Unfortunately, the answer is, it depends. It depends on how much effort you can muster up. According to some high intensity advocates, one set is all that’s required or pretty close to it. My question is, how many people can take one set to all-out, “as if your life depended on it” absolute failure, where the muscle can’t contract for one more millisecond? In my 20+ years of experience, I have found that not many can. You usually need at least 2-5 sets to really create a deep muscle burn. For people with low pain threshold, the super intensity approach is out of the question. It may require them a few more sets because with lower training intensity you need a bit more volume.

    Ultimately, training experience is your best guide. Try this method. After you’ve reached your peak weight and maximum number of reps for a specific movement, see if you can perform another set exactly the same way. If you can, then your initial effort wasn’t high enough and the next time you train either go heavier or perform more reps. If you can’t, don’t waste energy doing any more sets. You’ve provided an optimum growth stimulus and you should move on to the next exercise. If you’re goal is to train as intensely as possible, this method will give you a way to gauge your performance.

  13. The Importance of Frequent Meals

    When you skip a meal the body goes into a “fasting mode”. During this fasting, carbohydrates, protein and fats are all used for energy. Carbohydrates are the number one source of fuel for the body, followed by protein and then fat. As this fasting phase continues the following things happen:

    The muscle glycogen storage deposits in muscle and liver become depleted. As the glucose levels drop the body looks for other sources of fuel. Since the brain and nerves prefer energy from glucose, protein tissues (muscle) are broken down. (Not good if your aim is to build muscle). The effort to preserve the remaining protein, the body starts to metabolise fat for energy in the form of ketone bodies (acetoacetic acid). Decrease in T3 thyroid hormone, the longer the period between meals the greater the reduction inT3. Negative effect on insulin levels which causes increased insulin spikes which in turn leads to fat storage.
    The process of using ketone bodies as fuel is called ketosis. This process can cause the following negative effects on the body:

    • Nausea
    • Fatigue
    • Constipation
    • Lowered blood pressure
    • Elevated risks of kidney disease (uric acid)
    • Stale breath
    • Fetal harm/stillbirth in pregnant women.

    As the body switches to the use of ketone bodies, it begins to reduce energy output in an effort to protect both fat and lean tissue. (Lowering, of the metabolism). In addition, as the lean tissues begin to shrink, they become weaker and perform less metabolic work, reducing energy expenditure even more. Hormones also slow down the metabolism to conserve the lean body tissue. This is known as “starvation mode”, this mechanism is thought to have evolved as a defense mechanism against starvation. The body uses the calories efficiently in order to protect its fat stores using lean tissue and muscle instead. A lowered metabolic rate is a direct result of muscle loss. Accordingly; fewer calories are needed and weight loss slows down.

    Skipping meals and restricting calories will create cravings. Rapid high blood sugar occurs when you give into these cravings. As a result, your body creates a high amount of triglycerides, which convert to fat-storage. In addition, the depleted glycogen causes low energy levels because blood sugar is not being replaced. Unfortunately, the weight that is lost is mostly water (glycogen is made up of mostly water) and muscle.

    The slowed metabolism will also cause a slowdown digesting your food, if you cram all your food into one meal. This is also commonly seen in people who skip meals. They get so hungry they eat more than one meals worth of calories at one sitting. The already slowed metabolism is going to take a lot longer to digest the oversized meal through your digestive system again resulting in weight gain.

    The success rate of losing weight from skipping meals is poor. A significant amount of weight may be lost at first, but once again this is water and muscle weight. And you will in most instances put the weight back on plus more.

  14. Increase Your Testosterone Efficiency With Clomid

    When a bodybuilder uses a steroid cycle, he feels like Superman. His strength and muscle mass skyrocket, and his sex drive goes through the roof. He is aggressive and powerful. Once the cycle ends, however, it is quite another story. Whether it be from normal scheduled stops, or because the athlete simply runs out of drugs, there comes a time when testosterone supplementation has to stop. Very often, the endocrine system is damaged, and has halted production of testosterone by the testes due to the presence of artificial testosterone in the system for so long.

    When this happens, the bodybuilder has three options. First, he can get right back on the ‘sauce’ and start a new cycle. This is dangerous, as it only perpetuates the problem of endocrine shutdown. Also, it doesn’t give organs like the liver, kidneys, and prostate a chance to recover from the damage occurred while on cycle. The second option is to just lie back and suffer through the crash. Bodybuilder can lose 5 to 25 pounds of muscle when stopping a cycle cold turkey, and the effects upon the body, psyche, and life can be devastating. The endocrine system usually begins creating testosterone again, but it can be months or years before it has recovered fully. This can put your life, training, or family planning on hold.

    The third option to halting the stop-testosterone crash is to employ a compound to help the body regain testosterone production and combat the negative effects of an estrogen spike, which also accommodate stopping testosterone supplementation. Compounds such as Clomid (Clomiphene citrate) help the body to halt estrogen production. This in turn protects existing testosterone in the body (however low the levels) from being aromatized into estrogen.

    Clomid is also used as a fertility drug in women. The same function that occurs in men occurs in women, only reversed. Instead of inspiring the hypothalamus to continue or restart production of testosterone, it encourages the estradiol levels to rise to encourage higher rates of fertility.

    Clomid is becoming more and more popular among athletes, scientists, and doctors alike. Unlike any of the back door drugs in bodybuilding such as DNP and insuling, studies have been conducted on athletes to determine the efficacy of Clomid in boosting or restoring testosterone production in men. If you need a boost, check the web or with your doctor. Clomid might provide the boost you?re seeking to keep testosterone levels high following a cycle.

  15. Muscle Steroid

    The human body is an amazing thing. Without even really knowing why, the average man or woman can perform amazing feats that scientists have worked for years to replicate in robots. The human body can run, jump, increase in strength and size, and even heal itself. And now that technology is so far advanced, new items are coming out all the time that are geared towards enhancing human performance in every area conceivable. One of the items that has been noted for truly enhancing bodily systems is the use of a muscle steroid.

    There are many different varieties that you can find if you are looking for a muscle steroid. There are some geared towards women, and some specifically designed for men. And depending on the results that you want to achieve, you can choose between a list of options, in order to find the best fit for you.

    A muscle steroid is generally produced in a laboratory, and consists of certain hormones or chemicals that will stimulate body processes. Because it is used to enhance human performance (in this case, build muscle mass and strength,) it may also be referred to as an anabolic steroid. The hormone used most often, perhaps, in steroids is the male sex hormone, testosterone.

    Testosterone is the hormone in a man’s body that gives him his strength, allows him to build muscle mass, and does other things, such as deepen his voice, and cause hair growth. As extra testosterone is added to the body, it stimulates those mechanisms that produce protein blocks, thus encouraging rapid growth of muscle. It will also cause the voice to deepen even further, and, in some men, even increase the size of their testicles.

    Many changes will occur with the use of a muscle steroid. However, caution is always given to those who choose to try them out for the first time. Besides all of the seemingly “good” side effects, there are those effects that are less desirable. Some of these may include baldness, too much hair growth in other places, high cholesterol and blood pressure, and can even lead to more serious issues such as liver or heart problems, or even sexual dysfunction and infertility. As with any other substance, one must understand and accept all the possible consequences of using a drug like a muscle steroid. But if the pros outweigh the cons, you may find that it does enhance your muscle size and strength.